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. Author manuscript; available in PMC: 2015 Feb 1.
Published in final edited form as: Transl Stroke Res. 2013 Nov 27;5(1):118–127. doi: 10.1007/s12975-013-0312-z

Figure 1. The profile of HIF-1α, HIF-2α, and PHD-2 following 2 hours of OGD.

Figure 1

(A) A representative image of a western blot of HIF-1α, HIF-2α, PHD-2 and the β-actin as the loading control (B) Densiometric quantification of HIF-1α expression. (C) HIF-2α expression following EPO treatment. (D) PHD-2 expression following OGD. Data represent mean (± SD) n=6 in all groups. *P<0.01 compared to normoxia. δP< 0.01 compared to untreated. #P< 0.01 compared to 10U/ml EPO treated group. One way ANOVA was used for all analysis. HIF-1α, P ≤ 0.001 using Kruskal-Wallis ANOVA on Ranks. PHD-2 P < 0.001. HIF-1α but not HIF-2α expression was altered in the presence of EPO treatment.