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. Author manuscript; available in PMC: 2015 Feb 1.
Published in final edited form as: J Nutr Biochem. 2013 Nov 6;25(2):126–135. doi: 10.1016/j.jnutbio.2013.10.003

Fig. 7.

Fig. 7

Relative mRNA levels of inflammatory and xenobiotic-related markers, Cyp1A1 (A), Cyp1B1 (B), MCP-1 (C) and CCL3 (D) in mouse liver samples. GTE supplementation led to increased cytochrome P450 (CYP1A1 and CYP1B1) mRNA expression in the presence of both GTE and environmental toxicant (i.e., PCB 126), indicating increased activity for toxicant degradation and/or excretion. MCP-1 and CCL3 inflammatory markers were statistically increased in GTE-supplemented mice liver samples but toxicant-induced inflammatory markers returned to control levels due to GTE supplementation. All values were determined using the relative quantification method (ΔΔCt) as a fold change from control. Data are presented as mean±S.E.M. (*p<0.01, **p<0.05, n=8–10).