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. Author manuscript; available in PMC: 2015 Apr 1.
Published in final edited form as: Neuropharmacology. 2013 Dec 4;79:249–261. doi: 10.1016/j.neuropharm.2013.11.022

Fig. 9.

Fig. 9

Glutamate-induced neuronal injury is only partially dependent on AT1A receptors in mouse CGCs. Primary CGCs from wild-type (A) or AT1A knock-out (B) mice were exposed to glutamate (Glut) (100 µM) with or without telmisartan (Telm) (1 µM). Treatment with telmisartan significantly reduced glutamate-induced LDH release from CGCs obtained from wild-type (A) or AT1A knock-out (B) mice. Results are Means ± SEM for groups of three independent experiments. *P < 0.05, **P < 0.01, ***P < 0.001 vs. untreated controls; #P < 0.05, ##P < 0.01, ###P < 0.001 Glut vs. Glut+Telm.