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. Author manuscript; available in PMC: 2015 Feb 1.
Published in final edited form as: Am J Obstet Gynecol. 2013 Sep 20;210(2):154.e1–154.e8. doi: 10.1016/j.ajog.2013.09.030

Figure 3.

Figure 3

Decreasing ANTXR2 expression on human uterine smooth muscle cells (HUSMC) resulted in decreased oxytocin-induced HUSMC contractility. A) Images of detached collagen lattices 48h after addition of 0nM or 100nM oxytocin. B) HUSMC with decreased ANTXR2 expression (HUSMC-R2KD) did not contract the collagen gel well after oxytocin administration, resulting in collagen gel surface areas similar to control (HUSMC-CTL) cells that were not treated with oxytocin. HUSMC with increased ANTXR2 expression (HUSMC-R2OE) did not contract any better than and HUSMC-CTL cells treated with oxytocin.