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. 2014 Mar 14;9(3):e91512. doi: 10.1371/journal.pone.0091512

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© 2014 Feng et al

This is an open-access article distributed under the terms of the Creative Commons Attribution License, which permits unrestricted use, distribution, and reproduction in any medium, provided the original author and source are properly credited.

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The NPY+ interneurons were evenly distributed in the striatum in the control group (A and A′). This interneuron type in QA-treated rats (B and B′) hardly survived in the lesion core (★) and experienced some changes in the transition zone (*) including decrease in neuron number and increase in the number of varicosities which formed along the neuronal processes (see the arrow in B′ and the view of higher magnification in B*), but hyperplasia of fibers was not obvious. The ChAT+ interneurons were large in size and evenly distributed throughout the striatum in the control rats (C and C′) and they appeared stable in neuron abundance as well as in morphology without hyperplasia of fibers or varicosity formation in the QA-treated rats (D and D′). Panels A′–D′ are views of higher magnification from the boxes of Panels A–D, respectively. Cont is short for control. Scale bars: A–D, 100 μm; A′–D′, 30 μm; B*, 10 μm.