Figure 3.
After HDC knockdown in cholangiocarcinoma cells (Mz-HDC), xenograft tumor development was significantly inhibited when compared to the tumors containing normal levels of HDC (Mz-neg). Further, in tumor sections extracted from the animal model (that contain 99% cholangiocarcinoma cells), both proliferating cellular nuclear antigen and HDC expression were significantly decreased in the HDC knockdown transfected tumors (Mz-HDC). (Reprinted with permission from Francis et al. Gut, 2012;61:753-64.)