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. 2013 Dec 10;18(3):455–467. doi: 10.1111/jcmm.12196

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© 2013 The Authors. Journal of Cellular and Molecular Medicine published by John Wiley & Sons Ltd and Foundation for Cellular and Molecular Medicine.

This is an open access article under the terms of the Creative Commons Attribution License, which permits use, distribution and reproduction in any medium, provided the original work is properly cited.

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Increase in uterine at-RA level and decrease in its synthase levels during mice peri-implantation following pCR3.1-cyp26a1 immunization, especially on D5 of mice pregnancy. (A) high performance liquid chromatography (HPLC) chromatographs of RA. 1 for 13-cis-RA; 2 for 9-cis-RA; 3 for at-RA. (B) The HPLC chromatograms of D5 uterine at-RA. (C) Uterine at-RA concentration (ng/g) increased in uterus based on uterine at-RA concentration (ng/g) trend graph, under the influence of recombinant plasmid. The uterine at-RA concentration (ng/g) was evaluated by HPLC analysis. Pairwise comparisons were taken between each treatment group, *P < 0.05. At least, five independent experiments were repeated for each time-point. A total of 74 mice were assessed. (D) RALDH1A1 and RALDH1A2 levels were significantly reduced at uterine implantation sites based on Western blotting of pregnancy D5. β-actin was used as a loading control.