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. 2014 Mar 17;9(3):e91890. doi: 10.1371/journal.pone.0091890

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© 2014 Giachini et al

This is an open-access article distributed under the terms of the Creative Commons Attribution License, which permits unrestricted use, distribution, and reproduction in any medium, provided the original author and source are properly credited.

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Concentration-response curves to (A) ACh (without NOS and COX inhibitors incubation) or (B) SNP in the presence of L-NAME (100 μM) plus indomethacin (10 μM) in arteries from (○) vehicle–treated control (n = 5); (•) clopidogrel-treated control (n = 5); (□) vehicle-treated Ang II-hypertensive (n = 6); and (▪) clopidogrel-treated Ang II hypertensive-rats (n = 6). Experimental values of the relaxation induced by ACh were calculated relative to the maximal changes from the contraction produced by PE in each tissue, which was taken as 100% (% of relaxation). Results are presented as mean ± SEM in each experimental group. *, P<0.05 vs. vehicle-treated control rats; †, P<0.05 vs. vehicle-treated Ang II rats.