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. 2014 Mar 18;5:60. doi: 10.3389/fpls.2014.00060

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Copyright © 2014 Park, Jeong and Kim.

This is an open-access article distributed under the terms of the Creative Commons Attribution License (CC BY). The use, distribution or reproduction in other forums is permitted, provided the original author(s) or licensor are credited and that the original publication in this journal is cited, in accordance with accepted academic practice. No use, distribution or reproduction is permitted which does not comply with these terms.

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The conversion of potexvirus virions into translatable vRNA. PVX TGB1 protein binds to polar CP subunits located at the end of PVX virion (A,B). Then, binding of TGB1 protein causes the CP to separate from the PVX virion (C) early in infection after movement of virion into uninfected adjacent cells, such that the virion become translatable. Replicase then replicates (-) vRNA from the translatable form of PVX (+) vRNA (D), replicase transcribed (-) vRNA from (+) vRNA, and finally (not shown), ribosomes bind to the (+) vRNA.