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(a) OGD for 10 min caused a significant increase in CaMKII activity (1.43 ± 0.13, n = 4, P < 0.05). (b) OGD treatment for 25 min led to inhibition of CaMKII activity (0.65 ± 0.07, n = 3, P < 0.05). (c) 1 hr after PT, CaMKII activity displayed an upward trend (1.38 ± 0.07, n = 3, P < 0.05). (d) 12 hr after PT, CaMKII activity was inhibited (0.41 ± 0.19, n = 3, P < 0.05).
