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. 2014 Jan 30;65(6):1455–1465. doi: 10.1093/jxb/eru003

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© The Author 2014. Published by Oxford University Press on behalf of the Society for Experimental Biology.

This is an Open Access article distributed under the terms of the Creative Commons Attribution License (http://creativecommons.org/licenses/by/3.0/), which permits unrestricted reuse, distribution, and reproduction in any medium, provided the original work is properly cited.

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Fig. 2. — Expression of the BPC1–EAR chimeric protein causes strong developmental defects. (A) A 35S::BPC1–EAR plant with wild-type phenotype. (B) A 35S::BPC1–EAR plant with a severe phenotype. Leaves are small and curled (arrowhead). (C) Leaf of a 35S::BPC1–EAR plants with both abaxial and adaxial sides covered by trichomes. (D) Inflorescence of a 35S::BPC1–EAR plant with a severe phenotype. Flowers displayed petaloid sepals (arrowhead). (E) A pALC::BPC1–EAR plant after 6 d of ethanol induction in which the IM arrested prematurely (arrowhead). (F) A flower of a pALC::BPC1–EAR plant after 4 d of ethanol induction with petaloid sepals (arrowhead), no petals, and short and aberrant stamens. Pi, pistil; Se, sepal; St, stamen; Ps, petaloid sepals; Up, upper side; Lo, lower side. (This figure is available in colour at JXB online.)