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. 2014 Jan 6;2(1):e00196. doi: 10.1002/phy2.196

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© 2014 The Authors. Physiological Reports published by Wiley Periodicals, Inc. on behalf of the American Physiological Society and The Physiological Society.

This is an open access article under the terms of the Creative Commons Attribution License, which permits use, distribution and reproduction in any medium, provided the original work is properly cited.

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Figure 2. — Calcium‐independent phospholipase A₂ (iPLA₂) activity (A), arachidonic acid release (B), platelet‐activating factor (PAF) production (C), and adherence of polymorphonuclear leukocytes (PMN) (D) in human coronary artery endothelial cells infected with Trypanosoma cruzi (MOI 0.2) for up to 96 h. Cells were pretreated with 0.5 μmol/L (R)‐bromoenol lactone (BEL) or (S)‐BEL (10 min) prior to infection with T. cruzi. Values shown are means ± SEM for four separate cell cultures. **P < 0.01 when compared with uninfected controls. ⁺⁺P < 0.01 when comparing values in the presence or absence of BEL.