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. Author manuscript; available in PMC: 2015 May 1.
Published in final edited form as: Psychol Med. 2013 Sep 4;44(7):1499–1509. doi: 10.1017/S0033291713002286

Figure 1.

Figure 1

Panel A shows the bivariate model that was evaluated. The baseline bivariate model included genetic (A) and non-shared environment (E) latent variables that were common to combat and PTSD (A1 and E1) and A and E factors that were specific to PTSD (A2 and E2). All paths implicated in the etiology of PTSD were also moderated by combat exposure. Panel B shows the univariate model that was evaluated. This model included regressive paths from combat exposure in both twins to PTSD in twin 1 (the complimentary model for twin 2 is not show in the figure). The model also includes A and E effects for PTSD and moderation of these effects by combat exposure. In both types of models the cross-twin correlation between the A factors was set to 1.0 and .50 for MZ and DZ twins, respectively, and the cross-twin correlation between the E factors was set to 0. M = moderator; sx = symptoms.