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. 2014 Feb 18;289(14):9741–9753. doi: 10.1074/jbc.M113.517995

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© 2014 by The American Society for Biochemistry and Molecular Biology, Inc.

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FIGURE 1. — The endogenous H₂S pathway was down-regulated in ox-LDL-induced, THP-1-derived macrophages (mean ± S. D.). A, changes of endogenous H₂S-generating enzymes in ox-LDL-induced, THP-1-derived macrophages (mean ± S.D.). The H₂S level was analyzed by sensitive sulfite electrode. *, p < 0.05 compared with a control group. B, representative fluorescent staining (blue) of endogenous H₂S in THP-1-derived macrophages treated with ox-LDL for various time periods. C and D, CBS protein (C, analysis by Western blotting) and mRNA (D, analysis by real-time PCR) expressions decreased in ox-LDL-induced, THP-1-derived macrophages. *, p < 0.05 compared with a control group. E, mRNA and protein expression of CSE in THP-1-derived macrophages after ox-LDL treatment for 12 and 24 h. F, mRNA and protein level of MPST in THP-1-derived macrophages after ox-LDL treatment for 12 and 24 h. Data are means ± S.D. of three independent experiments performed in triplicate.