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. 2014 Mar;22(2):136–142. doi: 10.4062/biomolther.2014.006

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Copyright © 2014 The Korean Society of Applied Pharmacology

This is an Open Access article distributed under the terms of the Creative Commons Attribution Non-Commercial License (http://creativecommons.org/licenses/by-nc/3.0/) which permits unrestricted non-commercial use, distribution, and reproduction in any medium, provided the original work is properly cited.

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Fig. 3. — CGA protects cells against UVB-induced DNA damage. (A) DNA damage was assessed in an alkaline comet assay. Representative images and the percentage of total DNA fluorescence in the comet tails are shown. ^*Significantly different from control cells (p<0.05); ^#significantly different from UVB-irradiated cells (p<0.05). (B) HaCaT cells were treated with CGA. After 1 h, the cells were exposed to UVB radiation and cell viability was determined 20 h later in an MTT assay. ^*Significantly different from control cells (p<0.05); ^#significantly different from UVB-irradiated cells (p<0.05).