Skip to main content
. 2014 Mar 26;2014:bcr2013202939. doi: 10.1136/bcr-2013-202939

Figure 1.

Figure 1

(A) Cranial MRI in January 2010 revealed lesion of the periaqueductal grey with extension to the left cerebellar hemisphere as well as to the pons and the medulla oblongata on T2-weighted images, after a new attack presenting as bilateral internuclear ophthalmoplegia, crossed brainstem symptoms, nausea and intractable hiccups. (B) Spinal MRI in July 2010 showed a newly occurred T2 hyperintense lesion that affected almost the entire cross-section of the medulla oblongata and the pons, causative for a new relapse with brainstem symptoms. (C) T2-weighted spinal cord MRI in July 2012, when the patient presented with sensory disturbances, revealed multiple circumscribed hyperintense lesions, the cranial one extending over two vertebral segments. (D) Spinal cord MRI in October 2012 after a severe clinical deterioration to an Expanded Disability Status Scale of 9 demonstrated an extensive myelopathy from cervical vertebra 1 to 7. (E) MRI in August 2013 showed an almost complete restitution of the previously described myelopathy on T2-weighted images, after 10 months treated with tocilizumab.