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. 2014 Apr 7;9(4):e93853. doi: 10.1371/journal.pone.0093853

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© 2014 Park et al

This is an open-access article distributed under the terms of the Creative Commons Attribution License, which permits unrestricted use, distribution, and reproduction in any medium, provided the original author and source are properly credited.

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(A) Scheme of the competitive repopulation assay using GW1929, a high agonist of Pparg. (B) Analysis of the proportion of donor-derived HSCs obtained from untreated (Control) and treated (GW1929) recipient mice at 20 weeks posttransplant. Each dot represents a single mouse. (C) Relative contributions of or (T-cell lineage), (B-cell lineage) and Mac-1⁺ or Gr-1⁺ (myeloid lineage) cells in donor-derived Ly-5.1⁺ cells of recipient mice at 20 weeks posttransplant. Black, T-cell lineage; gray, B-cell lineage; white, myeloid lineage. Data are presented as the mean .

Inline graphic — (A) Scheme of the competitive repopulation assay using GW1929, a high agonist of Pparg. (B) Analysis of the proportion of donor-derived HSCs obtained from untreated (Control) and treated (GW1929) recipient mice at 20 weeks posttransplant. Each dot represents a single mouse. (C) Relative contributions of or (T-cell lineage), (B-cell lineage) and Mac-1⁺ or Gr-1⁺ (myeloid lineage) cells in donor-derived Ly-5.1⁺ cells of recipient mice at 20 weeks posttransplant. Black, T-cell lineage; gray, B-cell lineage; white, myeloid lineage. Data are presented as the mean .