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. 2014 Apr 7;9(4):e93563. doi: 10.1371/journal.pone.0093563

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© 2014 Jiang et al

This is an open-access article distributed under the terms of the Creative Commons Attribution License, which permits unrestricted use, distribution, and reproduction in any medium, provided the original author and source are properly credited.

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Uninjured kidneys showed no Pax2 and KIM-1 expression in proximal tubules. In IRI injured kidneys, there was 77.0±12.5% of Pax2-positive TECs co-expressed KIM-1(*P<0.05), indicating that injury induced proximal Pax2 was primarily expressed in injured proximal TECs.