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. Author manuscript; available in PMC: 2015 Apr 1.
Published in final edited form as: J Neurochem. 2013 Dec 13;129(2):315–327. doi: 10.1111/jnc.12616

Figure 2. Increased DA release from α6L9’S NAc.

Figure 2

A) Peak oxidative current vs. time is plotted for DA release responses from nonTg and α6L9’S NAc slices. Slices were stimulated with either 1 pulse (1P), or 4 pulses (4P) at the indicated stimulation frequency. The number of responses was: nonTg (1P, n=20; 4P 10 Hz, n=10; 4P 20 Hz, n=10; 4P 30 Hz, n=20; 4P 100 Hz, n=10), α6L9’S (1P, n=20; 4P 10 Hz, n=7; 4P 20 Hz, n=8; 4P 30 Hz, n=20; 4P 100 Hz, n=8).

B) Peak [DA]o versus stimulation frequency is shown for nonTg and α6L9’S NAc DA responses.

C) Relative DA signal (area under the peak oxidative current versus time curve) versus stimulation frequency is shown for nonTg and α6L9’S NAc DA release responses.

D) Extracellular DA levels over time are altered in α6L9’S NAc. For individual 1P and 4P (30 Hz) peak [DA]o vs. time responses in both nonTg and α6L9’S slices, decay tau values were derived by fitting the falling phase of the response to a single exponential decay function. ***p<0.001, **p<0.01