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. Author manuscript; available in PMC: 2015 Jan 1.
Published in final edited form as: Psychosom Med. 2013 Dec 23;76(1):20–28. doi: 10.1097/PSY.0000000000000025

Figure 3.

Figure 3

Acute TMT exposure induced an inflammatory phenotype in the heart at 24h by decreasing palmitoylethanolamide (PEA) and oleoylethanolamide (OEA) levels. Heart levels of: (A) PEA; (B) OEA; (C) N-acylphosphatidylethanolamine-specific phospholipase D (NAPE-PLD) activity; (D) N-acylethanolamine-hydrolyzing acid amidase (NAAA) activity; and (E) the ratio NAPE-PLD to NAAA activity. mRNA expression of (F) NAPE-PLD1 and (G) NAAA in heart with (H) ratio NAPE-PLD1 to NAAA mRNA. Data are expressed as mean ± s.e.m. (n = 10-12). * p<0.05; ** p<0.01.