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. Author manuscript; available in PMC: 2014 Oct 1.
Published in final edited form as: Nat Neurosci. 2014 Feb 23;17(4):559–568. doi: 10.1038/nn.3660

Figure 8. Partial effect of APL-specific RNAi of GABA biosynthesis.

Figure 8

(a–d) See grid at bottom for full genotypes. (a,b) α′ (a) and α lobe (b) responses to IA:EB mixtures (averages of responses to 1:4 and 4:1). n, left to right, given as number of brain hemispheres [number of flies]: 7 [5], 12 [8], 11 [10], 15 [12], 7, 6, 6, 6. (c,d) Population sparseness (c) and correlations of cell body responses to IA:EB mixtures (averages of responses to 1:4 and 4:1). n, left to right, given as number of brain hemispheres [number of flies]: 11 [9] (10 [9] in (d), 21 [14], 11 [10], 15 [12], 10, 10, 6, 6. (e) Sample activity maps of cell body responses analyzed in panels c and d. Compare to Fig. 5b. Scale bars, 10 μm. * P<0.05, ** P<0.01, *** P<0.001 significant difference between colored bars and relevant controls (gray bars), by unpaired Welch t-test for APL>shits1 and APL>GADRNAi (Mann-Whitney U test for APL>shits1 in d), and by one-way ANOVA and Dunnett’s multiple comparisons test for GH146-GAL4 and NP2631-GAL4 driving GADRNAi. § P<0.05 significant difference between effects of GADRNAi and APL>shits1 by 2-way ANOVA. Error bars show s.e.m.