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Relaxatory actions of estrogenic compounds are partly endothelial- and NO-dependent in human myometrial arteries. The relaxation of preconstricted myometrial arteries by 17β-estradiol (A), PPT (B) or DPN (C) was blunted following endothelial abrasion (open symbols, dashed lines) or by pretreatment with nitric oxide synthase inhibitor (L-NNA) (open symbols, solid lines). *Significant differences from intact artery responses. Data are presented as mean ± SEM (n = 6).
