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. Author manuscript; available in PMC: 2014 May 1.
Published in final edited form as: Stat Med. 2012 Sep 17;32(9):1494–1508. doi: 10.1002/sim.5613

Table III.

Analysis of the secondary trait prostate specific antigen in the case–control study of prostate cancer: test of no genetic effect.

CHR SNP γ01γ1 Test 1 (×10−4) Test 2 (×10−4) Test 3 (×10−4) MinT (×10−4)
2 rs10197545 0.704 0.953
0.953
0.552
0.400
0.729
0.460
0.953
3 rs17034929 −0.037 2.20
2.20
0.467
0.674
0.150
0.446
2.20
6 rs9448387 0.687 1.40
1.40
0.93
0.449
1.33
0.480
1.40
10 rs2255684 0.550 0.421
0.421
0.479
0.478
1.00
0.590
1.00
12 rs7135149 −0.150 1.46
1.46
0.675
1.50
0.465
1.44
1.46
12 rs11068651 0.414 0.600
0.600
0.246
0.250
0.199
0.240
0.600
13 rs9594159 −0.022 7.41
7.41
1.98
3.91
0.776
2.40
7.41
13 rs9316673 0.462 0.215
0.215
0.048
0.054
0.025
0.040
0.215
19 rs350134 0.095 4.06
4.06
1.66
2.20
0.990
1.90
4.06

CHR, Chromosome. SNP, single nucleotide polymorphism; Test 1, p-value of the Wald test assuming τ2 = 0; Test 2, p-value of the Wald test assuming τ2 = 0.05 × 0.95; Test 3, p-value of the Wald test assuming τ2 = 0.1 × 0.9. MinT, p-value of the minimum test for the baseline prevalence rate ranging from 0% to 10% evaluated at an interval of 0.2%. The second entry is the maximum likelihood approach.