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The kallikrein/kinin system reduces apoptosis by inhibiting the caspase-related signaling pathways. In non-FBS medium, LPS exposure significantly increased the activity of caspase 3 compared to the negative control group. Treatment with rAAV-HK and kinin, but not rAAV-lacZ, inhibited the effects of LPS on caspase 3 activity (P<0.05). Kinin+Hoe140 had no effects on the LPS-induced increase in caspase 3 activity.
