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. 2014 May 1;4:9. doi: 10.1186/2045-9912-4-9

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Copyright © 2014 Mangus et al.; licensee BioMed Central Ltd.

This is an Open Access article distributed under the terms of the Creative Commons Attribution License (http://creativecommons.org/licenses/by/2.0), which permits unrestricted use, distribution, and reproduction in any medium, provided the original work is properly credited. The Creative Commons Public Domain Dedication waiver (http://creativecommons.org/publicdomain/zero/1.0/) applies to the data made available in this article, unless otherwise stated.

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Pathophysiology and possible mechanisms of protection following cardiac arrest. A: Comprehensive cascades of pathophysiology constituting global brain hypoxic ischemia and reperfusion injury following cardiac arrest (CA) and return of spontaneous circulation (ROSC). B: The possible mechanisms of protection investigated in literature included in this review were separated into three broad mechanistic categories. The effects of these interventions could decrease global brain injury after resuscitation from cardiac arrest and thus potentially improve survival and neurologic outcome.