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. Author manuscript; available in PMC: 2014 Sep 27.
Published in final edited form as: Nature. 2014 Jan 19;507(7493):513–518. doi: 10.1038/nature12910

Extended Data Figure 5. Coordinated function of Ascl2 and Batf/IRF4 in regulating Tfh-related genes.

Extended Data Figure 5

(ChIP-Seq data of Maf, Batf, and IRF4 were derived from GSE40918 25)

a. Venn diagram of ChIP-Seq peaks from Ascl2 and Maf.

b. Distribution of ChIP-Seq peaks by Ascl2, Batf, and IRF4 on gene loci including Bcl6, Prmd1, Ascl2 and Maf.

c. Venn diagram of ChIP-Seq peaks from Ascl2, Batf and IRF4.

d. Distribution of ChIP-Seq peaks by Ascl2, Batf, and IRF4 on gene loci including Cxcr5, Cxcr4, Ccr7, Selplg1, Il2, Il2ra, and Il2rb. Blue frame represents the co-localization of peaks.

e. Distribution of ChIP-Seq peaks by Ascl2, Batf, and IRF4 on Th1-related Tbx21 and Ifng gene loci.

f. Distribution of ChIP-Seq peaks by Ascl2, Batf, and IRF4 on Th2-related Gata3, Il4, Il13 and Il5 gene loci.

g. Distribution of ChIP-Seq peaks by Ascl2, Batf, and IRF4 on Th17-related Rorc, Il17a, Il17f and Il21 gene loci.

ChIP-Seq assay of Ascl2 were performed on Ascl2-overexpressed T cell cultured under Th0 condition. ChIP-Seq assay of Maf, Batf and IRF4 were performed on Th0 cells by Dan R. Littman’s group, derived from GSE40918 25.