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. 2014 Apr 17;15(4):6556–6568. doi: 10.3390/ijms15046556

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© 2014 by the authors; licensee MDPI, Basel, Switzerland

This article is an open access article distributed under the terms and conditions of the Creative Commons Attribution license (http://creativecommons.org/licenses/by/3.0/).

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Figure 3. — (a) ARK1B10 expression level in non-tumorous liver tissue. HBV, positive for HBsAg; HCV, positive for anti-HCV; NBNC, negative for HBsAg and anti-HCV. The box encompasses the twenty-fifth through seventy-fifth percentiles, and the horizontal line through the middle of the box indicates the fiftieth percentile (median). The tenth and ninetieth percentiles are shown as whisker caps; and (b) Regression analysis of the relationship between AKR1B10 expression and hepatic steatosis in non-tumorous liver tissues.

Figure 3. — (a) ARK1B10 expression level in non-tumorous liver tissue. HBV, positive for HBsAg; HCV, positive for anti-HCV; NBNC, negative for HBsAg and anti-HCV. The box encompasses the twenty-fifth through seventy-fifth percentiles, and the horizontal line through the middle of the box indicates the fiftieth percentile (median). The tenth and ninetieth percentiles are shown as whisker caps; and (b) Regression analysis of the relationship between AKR1B10 expression and hepatic steatosis in non-tumorous liver tissues.