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. 2014 Apr 13;13:82. doi: 10.1186/1476-4598-13-82

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Copyright © 2014 Weng et al.; licensee BioMed Central Ltd.

This is an Open Access article distributed under the terms of the Creative Commons Attribution License (http://creativecommons.org/licenses/by/4.0), which permits unrestricted use, distribution, and reproduction in any medium, provided the original work is properly credited. The Creative Commons Public Domain Dedication waiver (http://creativecommons.org/publicdomain/zero/1.0/) applies to the data made available in this article, unless otherwise stated.

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HA–CD44 interaction increases Akt activation and promotes myxoma virus oncolysis in GBC cells in vitro. A. Western blotting results of p-Akt and total Akt expression in GBC cell lines (GBC-SD, SGC-996) or the glioma cell line (U251) pretreated with MYXV (Myx), Myx + Rap, Myx + HA, or Myx + HA + anti-CD44 mAb. B. Relative gray scale of p-Akt values from each group (*, P < 0.05 compared with the control group). C. Oncolytic effects of MYXV on gallbladder cancer cell lines in vitro. GBC-SD, SGC-996, and U251 were infected with control Dead Virus (DV), Myx, Myx + Rap, HA, Myx + HA, Myx + HA + anti-CD44 mAb, or Myx + HA + isotype mAb (*, P < 0.05 compared with the control group).