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. 2014 May 15;63(6):2063–2072. doi: 10.2337/db13-1279

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© 2014 by the American Diabetes Association.

Readers may use this article as long as the work is properly cited, the use is educational and not for profit, and the work is not altered. See http://creativecommons.org/licenses/by-nc-nd/3.0/ for details.

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A: Erlotinib treatment decreased kidney ER stress, as indicated by decreased glomerular and tubule epithelial expression of CHOP, PERK, and BIP in STZ-eNOS^−/− mice. B: LC3A immunostaining was detected in tubular epithelial cells, but not in glomerulus, in vehicle-treated STZ-eNOS^−/− mouse kidneys. With erlotinib treatment, LC3A expression was detectable in glomerulus and was markedly increased in tubular epithelial cells. Original magnification: CHOP and BIP, ×250; PERK and LC3A, ×400.