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Role of innate immune cells in the pathogenesis of Con A-induced acute liver injury. Following Con A administration, CCL25 expression is up-regulated in the inflamed liver and CCR9+ macrophages accumulate to this site, while pDCs are down-regulated. CCR9+ macrophages produce TNFα and promote proliferation of IFNγ-producing Th1 and NKT cells via TLR4/6.
