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DA administration produced subcellular renal damage. TEM of renal cell injury revealed vascular damage, including (A, arrow) endothelial cell swelling, (A, arrowhead) epithelial cell vacuolization, (B) proximal tubule cell detachment, and (C) luminal protenaceous material and cellular debris. Higher-magnification views more clearly showed (D, arrow) endothelial cell damage, (E) toxic vacuolization, and (F, arrows) mitochondrial swelling after DA administration.
