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. 2014 May 5;111(20):7438–7443. doi: 10.1073/pnas.1405289111

Fig. 1.

Fig. 1.

Power and variance of CGm signal in SCZ and BD. (A) Power of CGm signal in 90 SCZ patients (red) relative to 90 HCS (black) (see SI Appendix, Table S1 for demographics). (B) Mean power across all frequencies before and after GSR indicating an increase in SCZ [F(1, 178) = 7.42, P < 0.01], and attenuation by GSR [F(1, 178) = 5.37, P < 0.025]. (C) CGm variance also showed increases in SCZ [F(1, 178) = 7.25, P < 0.01] and GSR-induced reduction in SCZ [F(1, 178) = 5.25, P < 0.025]. (D–F) Independent SCZ sample (see SI Appendix, Table S2 for demographics), confirming increased CGm power [F(1, 143) = 9.2, P < 0.01] and variance [F(1, 143) = 9.25, P < 0.01] effects, but also the attenuating impact of GSR on power [F(1, 143) = 7.75, P < 0.01] and variance [F(1, 143) = 8.1, P < 0.01]. (G–I) Results for BD patients (n = 73) relative to matched HCS (see SI Appendix, Table S3 for demographics) did not reveal GSR effects observed in SCZ samples [F(1, 127) = 2.89, P = 0.092, n.s.] and no evidence for increase in CGm power or variance. All effects remained when examining all gray matter voxels (SI Appendix, Fig. S1). Error bars mark ± 1 SEM. ***P < 0.001 level of significance. n.s., not significant.