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. 2014 Feb 12;15(3):620–629. doi: 10.1208/s12249-014-0086-y

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Fig. 6 — Representative photographs of cross-sections of liver in mice with liver fibrosis induced by DMN (a) and in mice with liver fibrosis induced by CCl₄ (b) (A, healthy group; B, saline control group; C, empty liposome group; D, OMT-SOL group; E, OMT-LIP group). Black arrow, inflammatory cells; white arrow, necrotic hepatocytes; blue arrow, swollen hepatocytes; yellow arrow, fatty degeneration; red arrow, deposited collagens. Left panel Hematoxylin–eosin staining. Bar, 50 μm. Right panel Masson’s trichrome staining. Bar, 200 μm

Fig. 6 — Representative photographs of cross-sections of liver in mice with liver fibrosis induced by DMN (a) and in mice with liver fibrosis induced by CCl₄ (b) (A, healthy group; B, saline control group; C, empty liposome group; D, OMT-SOL group; E, OMT-LIP group). Black arrow, inflammatory cells; white arrow, necrotic hepatocytes; blue arrow, swollen hepatocytes; yellow arrow, fatty degeneration; red arrow, deposited collagens. Left panel Hematoxylin–eosin staining. Bar, 50 μm. Right panel Masson’s trichrome staining. Bar, 200 μm