Fig. 2.

Phenotypes of A. thaliana lfy mutants containing LFY transgenes. (a) IacLFY replaces endogenous AthLFY function, resulting in a wild-type architecture. (b–d) Compared with inflorescences from control AthLFY transformants (b), inflorescence elongation was inhibited in IscLFY1 transformants (c), and bracts (arrows) are often formed (d). (e and f) IscLFY1 lines showed modified floral architecture demonstrating an apetal1-like phenotype (e) and a reduction in petal number to an average of 1.38 ± 0.50, often showing complete apetaly (f). (g) Aerial rosettes are produced on some secondary inflorescence shoots of IscLFY1 transgenic plants (arrow). (h) The structure of one dissected rosette (Upper) is diagrammed (Lower) to show that each fruit/flower (yellow) has a subtending bract (green) indicated by a linking red line. (i) LcrLFY rescued the lfy mutation but caused flowers with 6.65 ± 1.27 petals rather than the typical four petals (sepal and stamen number is unchanged). (j) LcrLFY modifies inflorescence architecture in A. thaliana. (k) All of the inflorescences are prematurely terminated by partial, terminal flowers, resulting in the release of determinate axillary shoots from the rosette.