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Total and uncoupled mitochondrial respiration are inhibited by the synthetic Errα-inverse agonist XCT790. (A and B) C2C12 myotubes were infected with GFP or PGC-1α adenovirus and were treated with vehicle (0.1% DMSO) or 1 μM XCT790 for 2 days before total mitochondrial respiration (A) and uncoupled respiration (B) was determined. *, P < 0.05 in paired t test.
