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. 2014 Apr 24;306(11):G917–G928. doi: 10.1152/ajpgi.00422.2013

Table 1.

Experimental details, strengths, and limitations of models of NEC in rats

Experimental Details Strengths Limitations References
Formula feeding and hypoxia (newborn rats). Utilizes known clinical risk factors. Rats may be exposed to maternal breast milk before induction of the model. 7
Formula feeding, hypoxia and hypothermia (newborn rats). Utilizes known clinical risk factors. Rats may be exposed to maternal breast milk before induction of the model. 6
High disease severity.
Formula feeding, hypoxia and hypothermia (premature rats). No maternal milk exposure. Cesarean section is required. 14, 15, 28, 29, 47, 48, 53, 58, 59
Intestinal barrier is immature. In patients, NEC does not develop immediately after birth questioning the clinical relevance.
Formula-feeding, hypoxia and hypothermia with the addition of commensal bacteria from adult rats (premature rats). Includes risk factors associated with bacterial colonization. The bacterial components that are added may not be representative of the clinical situation. 13, 14, 25, 37, 66, 88
High disease severity.
Formula-feeding, hypoxia and hypothermia with the addition of intragastric LPS. The introduction of bacterial component (i.e., LPS). LPS is not representative of all bacterial components relevant to NEC. 21, 32, 38
Formula-feeding, hypoxia, hypothermia reoxygenation (100% O2 for 5 min). Increased intestinal injury. The delivery of 100% oxygen to premature infants is avoided given known toxicity. 62
Formula-feeding and hypoxia, reoxygenation (5% oxygen for 3 to 10 min), no hypothermia. Avoidance of hypothermia simplifies the model. Clinical relevance is uncertain given the prolonged reoxygenation. 19
Formula-feeding and hypoxia, addition of Cronobacter sakazakii. Allows for the direct investigation of the role of Cronobacter sakazakii, which is associated with NEC. Cronobacter sakazakii causes a minority of NEC cases in patients, and is more likely to be associated with other infections (i.e., meningitis). 52
Ischemia and reperfusion of the intestine in older rats. More accurately is an oxidative stress model. Performed in older animals when the intestine is more mature; relevance to clinical NEC is uncertain. 50, 80

NEC, necrotizing enterocolitis.