Skip to main content
. 2014 Mar 26;306(11):R804–R813. doi: 10.1152/ajpregu.00074.2014

Fig. 1.

Fig. 1.

A: representative examples of renal sympathetic nerve activity (SNA) and arterial blood pressure (ABP) responses to paraventricular nucleus (PVN) injection of N-methyl-d-aspartate (NMDA) (left) and α-amino-3-hydroxy-5-methyl-4-isoxazolepropionic acid (AMPA) (right) at baseline, 10 min post-, and 120 min postmicroinjection of (2R)-amino-5-phosphonopentanoate (AP5) and 6-cyano-7-nitroquinoxaline-2,3-dione (CNQX), respectively. B: summary data of peak changes of renal SNA and mean arterial pressure (MAP) in response to PVN NMDA (n = 5) and AMPA (n = 5) at baseline, 10 min post-, and 120 min postmicroinjection of AP5 and CNQX, respectively. Note that AP5 and CNQX nearly abolished responses to their respective receptor agonists NMDA and AMPA at 10 min. Responses recovered after 120 min. *P < 0.05 vs. baseline response.