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. 2014 Jun 2;211(6):1049–1062. doi: 10.1084/jem.20131751

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© 2014 Ahn et al.

This article is distributed under the terms of an Attribution–Noncommercial–Share Alike–No Mirror Sites license for the first six months after the publication date (see http://www.rupress.org/terms). After six months it is available under a Creative Commons License (Attribution–Noncommercial–Share Alike 3.0 Unported license, as described at http://creativecommons.org/licenses/by-nc-sa/3.0/).

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Figure 7. — Long-term treatment with RU-505 reduced the level of infiltrated fibrinogen and microgliosis in the cortex of Tg6799 mice. (A) Fibrinogen localized outside of endothelial cells of blood vessels was labeled with FITC-conjugated antifibrinogen antibody (green), and endothelial cells were labeled using anti-CD31 antibody (red; bars, 50 µm). (B) Activated microglia were visualized by staining for CD11b (red). DAPI staining (blue) was used to show integrity of tissue (bars, 100 µm). (C and D) Total fibrinogen area (C) and microgliosis (D) were quantified from 3 sections per mouse (n = 3–4 mice per group; *, P < 0.05; ***, P < 0.001). All values are means and SEM. Results are from two independent experiments.