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. 2014 May 3;10(5):543–549. doi: 10.7150/ijbs.8888

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© Ivyspring International Publisher. This is an open-access article distributed under the terms of the Creative Commons License (http://creativecommons.org/licenses/by-nc-nd/3.0/). Reproduction is permitted for personal, noncommercial use, provided that the article is in whole, unmodified, and properly cited.

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The working model of DLG5 in cell polarity maintenance and cancer development. A. in normal cell (DLG5^+/+ cell); B in DLG5^-/-cell; C. loss of DLG5 induces EMT.