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. 2014 Jun 7;20(21):6666–6670. doi: 10.3748/wjg.v20.i21.6666

Table 1.

Studies included in the meta-analysis

SNP Ref. Year Country Ethnicity AP
Control
P value for HWE Source of controls AP Control Assay
AA AB BB AA AB BB
Asp299Gly Hofner et al[11] 2006 Hungary Caucasian 84 7 1 64 7 2 0.01 PB 92 73 Taqman
Gao et al[10] 2007 China Asian 101 22 0 71 9 0 0.59 PB 123 80 PCR-RFLP
Zhang et al[9] 2008 China Asian 238 0 0 121 0 0 1.00 PB 238 121 PCR-RFLP
Takagi et al[8] 2009 Japan Asian 202 0 0 286 0 0 1.00 PB 202 286 PCR-RFLP
Chen et al[12] 2009 China Asian 64 15 0 47 6 0 0.76 PB 79 53 PCR-RFLP
Guenther et al[7] 2010 Germany, United States Caucasian A 991 G 51 A 725 G 45 PB 521 385 Taqman
Thr399Ile Hofner et al[11] 2006 Hungary Caucasian 85 6 1 64 7 2 0.01 PB 92 73 Taqman
Takagi et al[8] 2009 Japan Asian 202 0 0 286 0 0 1.00 PB 202 286 PCR-RFLP
Guenther et al[7] 2010 Germany, United States Caucasian C 977 T49 C 728 T 36 PB 521 385 Taqman

SNP: Single nucleotide polymorphisms; AP: Acute pancreatitis; PB: Population-based study; HWE: Hardy-Weinberg equilibrium in controls; PCR-RFLP: Polymerase chain reaction-restriction fragment length polymorphism.