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. Author manuscript; available in PMC: 2015 May 1.
Published in final edited form as: Virology. 2014 Apr 19;0:259–267. doi: 10.1016/j.virol.2014.04.008

Fig. 7.

Fig. 7

Effect of IL-17 deficiency on control of MAV-1 replication in lungs. A) Wild type (IL-17+/+) and IL-17−/− mice were infected i.n. with MAV-1 or mock infected with conditioned media and lungs were harvested at the indicated time points. B) IL-17+/+ and IL-17−/− mice were infected i.n. with MAV-1 and then rechallenged with MAV-1 28 days after the primary infection. Lungs were harvested at 7 days post rechallenge. DNA was extracted from lungs and qPCR was used to quantify MAV-1 genome copy number. DNA viral loads are expressed as copies of MAV-1 genome per 100 ng of input DNA. Individual circles represent values for individual mice and horizontal bars represent means for each group. Statistical comparisons were made using two-way ANOVA followed by Bonferroni’s multiple comparison tests.