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. Author manuscript; available in PMC: 2014 Jun 16.
Published in final edited form as: Radiat Res. 2013 Aug 29;180(4):367–375. doi: 10.1667/RR3111.1

FIG. 2.

FIG. 2

Role of gap junction communication in the propagation of stressful effects in human cells exposed to low-LET radiation or high-LET radiation. Confluent human skin fibroblasts were exposed to different types of microbeam in the absence (■) or presence (□) of the gap junction inhibitor 18-α-glycyrrhetinic acid (AGA) and holding in the confluent state for 4 h at 37°C then the cells were subcultured and assayed for micronucleus formation: Dose response for frequency of micronuclei formation in bystander NB1RGB cells (panel A: X rays, panel B: carbon ions, panel C: neon ions and panel D: argon ions) (*P < 0.05; ***P < 0.0005).