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. 2004 May 1;18(9):1060–1071. doi: 10.1101/gad.1189704

Table 2.

Reciprocal regulation of endothelial cell gene expression by Akt and FKHR

GenBank # Gene name Max fold change myrAkt/GFP Max fold change FKHR/GFP
BT006809 Fatty acid binding protein 4 (FABP4) 0.1 7.09
NM_001964 Early growth response 1 (EGR1) 0.12 7.12
X96719 C-type lectin superfamily member 2 (CLEC2) 0.16 8.06
NM_001147 Angiopoietin 2 (Ang-2) 0.18 17.01
NM_001901 Connective tissue growth factor (CTGF) 0.21 3.12
NM_007036 Endothelial cell-specific molecule 1 (ESM-1) 0.25 10.52
NM_080760 Dachshund homolog (Drosophila) 0.25 3.58
NM_021229 Protein with strong similarity to netrin 4 0.27 3.71
X07820 Matrix metalloproteinase 10 (MMP 10) 0.27 3.93
NM_003775 EDG8 0.32 3.48
NM_002962 Cytokine A2 13.07 0.11
NM_005630 Solute carrier family 21 8.65 0.26
NM_030817 Protein of unknown function 7.46 0.28
NM_006988 ADAMTS1 7.44 0.44
NM_139314 Angiopoietin-like 4 6.16 0.13
NM_198337 Insulin induced gene 1 5.91 0.22
NM_019058 HIF-1 responsive RTP801 5.91 0.14
NM_004609 Transcription factor 15 5.06 0.09

HUVECs were infected with adenoviruses encoding either GFP or myrAkt for 16, 20, or 24 h. RNA was isolated and subjected to microarray analysis. Genes whose expression was either induced greater than fivefold or repressed greater than threefold by Akt and that we had previously shown to be regulated in the opposite direction by FKHR are listed. The number reported for the myrAkt/GFP ratio indicates the maximum fold change for that gene during the time course of the experiment (16, 20, or 24 h).