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. Author manuscript; available in PMC: 2015 Mar 20.
Published in final edited form as: Toxicology. 2014 Jan 28;317:40–49. doi: 10.1016/j.tox.2014.01.006

Table 1.

Identification of proteins from spots with decreased abundance that contributed to the separation of hippocampus proteome profiles of offspring exposed to CSE throughout development (GD1-PD21 [PLS-DA model, VIP values (≥1.7)].

Spot# VIP Protein Identification GI Number MOWSE Score
12 2.09 ATP synthase, H+ transporting, mitochondrial F1 complex, alpha subunit, isoform 1 6680748 577
14 2.07 aldolase C, fructose-bisphosphate 60687506 865
16 1.98 proteasome (prosome, macropain) subunit, beta type 5 6755204 235
18 1.94 M2-type pyruvate kinase 1405933 369
25 1.87 vacuolar H+ATPase B2 17105370 243
28 1.86 aldolase C, fructose-bisphosphate 60687506 342
33 1.82 ATP synthase, H+ transporting, mitochondrial F1 complex, alpha subunit, isoform 1 6680748 620
35 1.80 dihydropyrimidinase-related protein 6714522 207
37 1.80 ATP synthase, H+ transporting, mitochondrial F1 complex, alpha subunit, isoform 1 6680748 980
38 1.79 heat shock protein (hsp60) precursor 56383 699
40 1.78 Protein disulfide-isomerase A3 1352384 260
41 1.77 aldhehyde dehydrogenase family 5, subfamily A1 27369748 368