Fig. 5.

Posterior whole body images of SAP scintigraphy scans of a patient with systemic monoclonal immunoglobulin type (AL) amyloidosis who presented with major liver involvement and proteinuria in 2005. He responded well to chemotherapy with substantial regression of amyloid by 2009 when his liver and renal function had returned to normal. The underlying plasma cell dyscrasia then gradually relapsed during 2009-11 leading to recurrence of proteinuria caused by reaccumulation of renal amyloid shown in the 2011 scan.