Table 2.
Results of the pain-related evoked potentials before and after consuming caffeine 400 mg or smoking a cigarette
|
Groups |
N2 |
N2 |
P2 |
P2 |
PPA
|
PPA |
|---|---|---|---|---|---|---|
| Before | After | Before | After | Before | After | |
| Coffeine |
182.60 ± 12.49 |
182.43 ± 11.24 |
131.78 ± 10.39 |
131.78 ± 9.60 |
52.03 ± 20.46 |
48.48 ± 22.64 |
| |
[165.90-211.70] |
[161.35-206.70] |
[108.90-52.80] |
[115.00-153.00] |
[19.70-128.30] |
[16.45-144.70 |
| Nicotine |
182.15 ± 12.49 |
176.89 ± 10.36*** |
131.35 ± 9.35 |
128.32 ± 7.93* |
51.55 ± 26.32 |
46.07 ± 22.63 |
| [158.30-205.40] | [161.30-199.10] | [155.75-150.40] | [113.00-141.70] | [10.40-117.30] | [13.70-84.45] |
Results are presented as mean ± standard deviations and range.
*p < 0.05 t-test paired samples.
**p < 0.005 t-test paired samples.
After smoking N2 and P2 latency are significantly reduced showing a facilitation of trigeminal pain processing at supraspinal level.