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. 2014 Jun 25;34(3):e00112. doi: 10.1042/BSR20140020

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© 2014 The author(s) has paid for this article to be freely available under the terms of the Creative Commons Attribution Licence (CC-BY)(http://creativecommons.org/licenses/by/3.0/) which permits unrestricted use, distribution and reproduction in any medium, provided the original work is properly cited.

This is an Open Access article distributed under the terms of the Creative Commons Attribution Licence (CC-BY) (http://creativecommons.org/licenses/by/3.0/) which permits unrestricted use, distribution and reproduction in any medium, provided the original work is properly cited.

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(A) The wild-type and mutated sequences of the two predicted miR-34a binding sites within the PDGFR-α3′ UTR and one predicted miR-34a binding site within the PDGFR-β3′ UTR are shown. (B) Co-transfection of miR-34a and the wild-type PDGFR-α/β 3′ UTR caused a significant decrease in luciferase activity compared with the controls. (C) Co-transfection of miR-34a and the mutant PDGFR-α/β3′ UTR plasmids (including all mutants) did not cause a decrease in luciferase activity. The data represent the mean±S.D.;*P<0.01 compared with the controls.