Table 5.
The study cohort aged 18–70
| MAP Coef (P) |
Interaction Coef (P) | Model r2 (P) | ||
|---|---|---|---|---|
| African American | White | |||
| Vglom (µm3 × 106) | 0.042 (<0.01) | 0.023 (0.43) | −0.635 (0.15) | 0.054 (<0.01) |
| Glomerulosclerosis | 0.126 (<0.001) | 0.088 (<0.001) | −0.914 (0.16) | 0.184 (0.01) |
| Cortical fibrosis | 0.001 (<0.001) | 0.002 (<0.001) | −0.005 (0.52) | 0.196 (<0.001) |
| Intimal thickening | 0.003 (<0.001) | 0.003 (<0.001) | −0.007 (0.54) | 0.341 (<0.001) |
Multivariate least squares regression showing the relationships of race, MAP and their interactions with the pathological features of hypertensive nephropathy.
Coefficients are presented for the non-interactive effects of MAP for Vglom, glomerulosclerosis (%), cortical fibrosis, and arterial intimal thickening and for the MAP race interactions. Model r2 is adjusted and reflects interactions.
MAP, mean arterial pressure (mmHg); Coef, coefficient; P, probability.