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A: inhibition of endothelial nitric oxide synthase with NG-nitro-l-arginine methyl ester (l-NAME; 10−4 mol/l) significantly reduced ACh-mediated vasodilation in isolated human adipose microvessels from individuals without cardiovascular disease (n = 4 vessels). B: time-control responses of ACh-mediated vasodilation (n = 7 vessels) were not significantly different between the first and second measurements. All values are plotted as means ± SE. *Significant difference (P < 0.05).
