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. Author manuscript; available in PMC: 2014 Jul 9.
Published in final edited form as: Exp Neurol. 2012 Feb 14;235(1):238–245. doi: 10.1016/j.expneurol.2012.01.027

Figure 4. In vivo AIH affects NF-κß signaling.

Figure 4

The same tissue from Fig. 3 was also interrogated for NF-κß protein expression via Western blot (A). C1-C4 indicates replicates of control (normoxic) animals; H1-H4 indicates replicates of AIH-treated animals. Expression of the NF-κß p65 subunit (A, top panel, band indicated with asterisk; quantitated in B) and p50 subunit (A, middle panel, quantitated in C) were downregulated in SVZ tissue immediately following AIH. In panels B and C the “*” indicates p<0.05 vs. normoxia tissues. Densitometry results are expressed as a ratio of the protein of interest to Cyclophilin A.